3 papers
National Cancer InstituteUS$1.2mNational Institute of Allergy and Infectious DiseasesUS$3.1mNational Institute on AgingUS$452.8k
Molecular mechanisms of neuronal cell death
National Institute on AgingUniv of Massachusetts Med Sch WorcesterUS$452.8k2026–2028
Role of the Cell Death Machinery in a Somatic Autoinflammatory Syndrome
National Institute of Allergy and Infectious DiseasesSloan-kettering Inst Can ResearchUS$3.1m2025–2029
Understanding the role of autophagy-regulated cell death in the escape from replicative crisis
National Cancer InstituteSalk Institute for Biological StudiesUS$606.8k2019–2025
Experiments in mice whose ovaries had been removed linked ceruloplasmin deficiency to higher bone mass at the tissue level.Experiments in mice whose ovaries had been removed linked ceruloplasmin deficiency to higher bone mass at the tissue level.
An experiment in twelve-month-old mice found better learning and memory alongside more immature nerve cells and greater insulation around nerve fibres.An experiment in twelve-month-old mice found better learning and memory alongside more immature nerve cells and greater insulation around nerve fibres.
A drug screen in human connective-tissue cells linked the selective killing effect to a protein that receives growth signals.A drug screen in human connective-tissue cells linked the selective killing effect to a protein that receives growth signals.
Erastin and RSL3 shortened survival dose-dependently, an effect rescued by ferrostatin-1, while altering iron and lipid profiles selectively in males.Erastin and RSL3 shortened survival dose-dependently, an effect rescued by ferrostatin-1, while altering iron and lipid profiles selectively in males.