Peripheral artery disease

Disease9 papers4 findings

  • Humans4
  • Animals1
  • In silico2

Effects

2

neurovascular coupling

no changein humans1

1 study
  1. Peripheral artery disease has no effect on neurovascular coupling in humans.

    Humansin older adults with symptomatic peripheral artery disease vs controls

    Cognitive Impairment and Cerebral Haemodynamics in Individuals with Symptomatic Peripheral Artery Disease · American journal of physiology. Heart and circulatory physiology · 29 Sep 2026

Cognitive function

downin humans1

1 study
  1. Peripheral artery disease impairs cognitive function in humans.

    Humansin older adults with symptomatic peripheral artery disease vs controls

    Cognitive Impairment and Cerebral Haemodynamics in Individuals with Symptomatic Peripheral Artery Disease · American journal of physiology. Heart and circulatory physiology · 29 Sep 2026

Associations

2

Lipid Accumulation Product

upin humans1OR 1.36

1 study
  1. Lipid Accumulation Product is associated with peripheral artery disease in humans (OR = 1.36, 95% CI 1.18-1.58).

    Humansadjusted for potential confounders

    Association between lipid accumulation product and the prevalence of peripheral artery disease: A cross-sectional study based on NHANES 1999‒2004 · Clinics (Sao Paulo, Brazil) · 28 Sep 2026

Lipid Accumulation Product →

C-reactive protein

upin humans1

1 study
  1. C-reactive protein raises the risk of peripheral artery disease in humans (HR per SD 1.22, 95% CI 1.19-1.26).

    Humansper SD13 yearsn = 468,266

    Risk factors for atherosclerosis diverge across vascular beds · American journal of preventive cardiology · 19 Jun 2026

C-reactive protein →

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American Journal of Preventive CardiologyHumans

Risk factors for atherosclerosis diverge across vascular beds

Across 468,266 adults followed for 13 years, genetics drove initial disease onset, whereas clinical and lifestyle factors dominated subsequent…Across 468,266 adults followed for 13 years, genetics drove initial disease onset, whereas clinical and lifestyle factors dominated subsequent polyvascular spread and mortality.