There are 7 papers on mitophagy in aging research: none in people yet, 5 in animals, 1 in cells. The newest story, Nov 2, 2026: “Worms lacking a regulatory protein lived longer on certain diets”.
56 new papers in 30 daysSee all 1,317
National Institute on Aging$2.4MNational Heart, Lung, and Blood Institute$4MJapan Society for the Promotion of Science¥29M
Florida International UniversityUniversity of Rochester$340K2026
LncRNA Mediated Mitophagy in Cardioprotection
American Heart AssociationUniversity of Virginia$100K2026–2027
Natural Science Foundation of Hunan ProvinceCentral South University2026
Natural Science Foundation of Hunan ProvinceHunan University of Chinese Medicine2025
Critical evaluation of mitophagy, mitochondrial dysfunction and senescence axis
Biotechnology and Biological Sciences Research CouncilNewcastle University2024–2028
SESTRIN 2 THE DYSFUNCTIONAL PIECE ORCHESTRATING MITOPHAGY IN THE HIPPOCAMPAL AGING
Agencia Nacional de Investigación y DesarrolloUniversidad San SebastiánCLP 103K2024–2027
Mitophagy in mammalian aging and longevity
Natural Sciences and Engineering Research CouncilUniversité du Québec a MontréalCA$47K2023–2024
The role of impaired mitophagy and mitochondrial dysfunction in glaucomatous neurodegeneration
National Eye InstituteUniversity of North Texas Hlth Sci Ctr$99.4K2021–2024
Mechanisms of defective mitophagy and cellular senescence in HIV associated COPD
National Heart, Lung, and Blood InstituteFlorida International University$469K2019–2025
Exercise-Induced Mitophagy In Hippocampal Neurons Against AD
National Institute on AgingUniversity of Virginia$565K2022–2023
Worm experiments traced the lifespan benefit to vitamin B12-dependent enzymes and a process that removes mitochondria, the cell’s energy-producing structures.Worm experiments traced the lifespan benefit to vitamin B12-dependent enzymes and a process that removes mitochondria, the cell’s energy-producing structures.
Experiments in mice whose ovaries had been removed found that increasing a mitochondrial protein eased signs of osteoporosis.Experiments in mice whose ovaries had been removed found that increasing a mitochondrial protein eased signs of osteoporosis.
Experiments in neurons made from healthy donors aged 11 to 91 found impaired removal of damaged mitochondria, the cells’ energy-producing structures.Experiments in neurons made from healthy donors aged 11 to 91 found impaired removal of damaged mitochondria, the cells’ energy-producing structures.