Cells
Loss of body-clock protein linked to more artery plaques in mice
Experiments linked deletion of a body-clock protein in brown fat cells to greater plaque formation in aging and young mice genetically prone to artery disease.Experiments linked deletion of a body-clock protein in brown fat cells to greater plaque formation in aging and young mice genetically prone to artery disease.
Experiments in aged male mice, one year, against controlsMechanisms
Limit: Ucp1-Cre is not PVAT-specific, so effects from other brown fat depots may confound results