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Tat-mtscs-fxn

Intervention

1 paper2 findings

C, W, M, H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.

Company ties: findings with an author at a company, or from a paper whose competing-interests statement declares ties to one.

  • 1 lab in 1 country
  • Funders: Ataxia UK and 3 more
  • 2 of 2 findings with company ties
  • Animals1

Outcomes

2

Succinate dehydrogenase

upin mice1

1 study

  1. Tat-mtscs-fxn increases succinate dehydrogenase in mice.

    “restore the activity of the succinate dehydrogenase and produce a significant lifespan increase.”

    Animalsin mice models of Friedreich ataxiamuscle

    Frataxin-deficient neurons and mice models of Friedreich ataxia are improved by TAT-MTScs-FXN treatment

    Journal of cellular and molecular medicine5 Oct 2017

More on Succinate dehydrogenase

Lifespan

upin mice1

1 study

  1. Tat-mtscs-fxn extends lifespan in mice.

    “restore the activity of the succinate dehydrogenase and produce a significant lifespan increase.”

    Animalsin mice models of Friedreich ataxia

    Frataxin-deficient neurons and mice models of Friedreich ataxia are improved by TAT-MTScs-FXN treatment

    Journal of cellular and molecular medicine5 Oct 2017

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