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Sod@hmuio-mntcpp nanoparticles

Intervention

1 paper3 findings

C, W, M, H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.

  • 1 lab in 1 country
  • Funder: National Natural Science Foundation of China
  • Animals1

Outcomes

3

Ferroptosis

downin cells1

1 study

  1. Sod@hmuio-mntcpp nanoparticles inhibit ferroptosis in cells.

    “the nanocomposite could inhibit fibroblast senescence and ferroptosis”

    Cellsin fibroblasts

    Mesoporous MOFs with ROS scavenging capacity for the alleviation of inflammation through inhibiting stimulator of interferon genes to promote diabetic wound healing

    Journal of nanobiotechnologyMay 13, 2024

More on Ferroptosis

Cellular senescence

downin cells1

1 study

  1. Sod@hmuio-mntcpp nanoparticles inhibit cellular senescence in cells.

    “the nanocomposite could inhibit fibroblast senescence and ferroptosis”

    Cellsin fibroblasts

    Mesoporous MOFs with ROS scavenging capacity for the alleviation of inflammation through inhibiting stimulator of interferon genes to promote diabetic wound healing

    Journal of nanobiotechnologyMay 13, 2024

More on Cellular senescence

Wound healing

upin animals1

1 study

  1. Sod@hmuio-mntcpp nanoparticles improve wound healing.

    “In vivo experiments demonstrated that the SOD@HMUiO-MnTCPP nanoparticles (S@M@H NPs) significantly accelerated diabetic wound healing.”

    Animalsin diabetic wounds

    Mesoporous MOFs with ROS scavenging capacity for the alleviation of inflammation through inhibiting stimulator of interferon genes to promote diabetic wound healing

    Journal of nanobiotechnologyMay 13, 2024

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