BETA-2-MICROGLOBULIN
- 1 lab in 1 country
- Funder: Capital Medical University
- Cells1
Outcomes
4Apoptosis
upin human cells1
1 study
Apoptosis
upin human cells1
BETA-2-MICROGLOBULIN increases apoptosis in human cells.
“In addition, β2m and CSE reduced BEAS-2B cell proliferation and increased apoptosis.”
Cellsin BEAS-2B cells48 hours
International journal of chronic obstructive pulmonary disease20 Jun 2026
Oxidative stress and ROS signalling
upin human cells1
1 study
Oxidative stress and ROS signalling
upin human cells1
BETA-2-MICROGLOBULIN increases oxidative stress and ROS signalling in human cells.
“β2m and CSE also decreased MMP, increased ROS and mtROS levels, and reduced OCR, indicating mitochondrial dysfunction.”
Cellsin BEAS-2B cells48 hours
International journal of chronic obstructive pulmonary disease20 Jun 2026
Mitochondrial membrane potential
downin human cells1
1 study
Mitochondrial membrane potential
downin human cells1
BETA-2-MICROGLOBULIN decreases mitochondrial membrane potential in human cells.
“β2m and CSE also decreased MMP, increased ROS and mtROS levels, and reduced OCR, indicating mitochondrial dysfunction.”
Cellsin BEAS-2B cells48 hours
International journal of chronic obstructive pulmonary disease20 Jun 2026
Cellular senescence
upin human cells1
1 study
Cellular senescence
upin human cells1
BETA-2-MICROGLOBULIN induces cellular senescence in human cells.
“β2m and CSE increased SA-β-gal staining in BEAS-2B cells, indicating enhanced cellular senescence.”
Cellsin BEAS-2B cells48 hours
International journal of chronic obstructive pulmonary disease20 Jun 2026