MIR196A1

1. MIR196A1 decreases TP53 in human cells.

   “activated prosurvival pathways (YAP, MAPK) and reduced p53 levels.”

   Cellsin human corneal endothelial cells

   Therapeutic Potential of miR-196a in Protecting Corneal Endothelial Cells From Senescence Through Mitochondrial and TGF-β Signaling Modulation

   Translational vision science & technology·Jun 1, 2026

More on TP53

1. MIR196A1 protects against cellular senescence in human cells.

   “miR-196a mitigated TGF-β-induced senescence by improving mitochondrial function, suppressing autophagy, and modulating TGF-β signaling components.”

   Cellsin TGF-β-treated human corneal endothelial cells

   Therapeutic Potential of miR-196a in Protecting Corneal Endothelial Cells From Senescence Through Mitochondrial and TGF-β Signaling Modulation

   Translational vision science & technology·Jun 1, 2026

More on Cellular senescence

1. MIR196A1 increases cell viability in human cells.

   “miR-196a overexpression enhanced cell viability, promoted cell cycle progression, and upregulated stemness markers”

   Cellsin human corneal endothelial cells

   Therapeutic Potential of miR-196a in Protecting Corneal Endothelial Cells From Senescence Through Mitochondrial and TGF-β Signaling Modulation

   Translational vision science & technology·Jun 1, 2026

1. Lung cancer decreases MIR196A1 in silico.

   “24, including hsa-mir-196a and hsa-mir-323b found to be downregulated”

   In silicolung cancerlung

   Analysis of Age-Specific Dysregulation of miRNAs in Lung Cancer Via Machine learning: Biomarker Identification and Therapeutic Implications in Patients Aged 60 and Above

   bioRxiv (Cold Spring Harbor Laboratory)·Feb 14, 2026Preprint

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