National Institute of Allergy and Infectious DiseasesUS$2.3mNational Institute on AgingUS$706.5kNational Institute of Arthritis and Musculoskeletal and Skin DiseasesUS$377.3k
TFEB and age-associated B cells in autoimmunity
National Institute of Arthritis and Musculoskeletal and Skin DiseasesMayo Clinic RochesterUS$377.3k2025–2027
Autoimmunity, calorie restriction, and core body temperature
National Institute of Allergy and Infectious DiseasesSan Diego Biomedical Research InstituteUS$541.5k2025–2027
Ontogenetic niche of B cells at the CNS borders in homeostasis, aging and autoimmunity
National Institute of Allergy and Infectious DiseasesWashington UniversityUS$561.1k2022–2027
National Institute on AgingUniversity of California, San FranciscoUS$45.6k2024–2026
National Institute on AgingNational Institute on AgingUS$182.2k
Across 4,825 adults followed for 14.8 years on average, thyroid antibodies were linked to cardiovascular risk in participants under 40 but not in those aged 40 and older.Across 4,825 adults followed for 14.8 years on average, thyroid antibodies were linked to cardiovascular risk in participants under 40 but not in those aged 40 and older.
Across 144 diseases, median AUC fell from 0.679 with 2,920 proteins to 0.637 when evaluated on 1,460 proteins without model refitting.Across 144 diseases, median AUC fell from 0.679 with 2,920 proteins to 0.637 when evaluated on 1,460 proteins without model refitting.
Loss of the cGAMP-degrading enzyme ENPP1 allows microglia-derived signaling molecules to trigger STING-dependent inflammation across multiple brain cell types.Loss of the cGAMP-degrading enzyme ENPP1 allows microglia-derived signaling molecules to trigger STING-dependent inflammation across multiple brain cell types.