2 papers
National Institute on AgingUS$8.7mNational Institute of Neurological Disorders and StrokeUS$13.2mNational Heart, Lung, and Blood InstituteUS$262.5k
Mechanisms of Fibrinogen-Mediated Cerebrovascular Pathology in Cerebral Amyloid Angiopathy
National Institute of Neurological Disorders and StrokeRutgers Biomedical and Health SciencesUS$1.9m2026–2029
In-vivo MRI-based prediction of cerebral amyloid angiopathy in community-based older adults
National Institute on AgingRush University Medical CenterUS$656.5k2026–2031
National Institute on AgingMassachusetts General HospitalUS$129.3k2026–2031
Endothelial Enhancer of Zeste Homolog 2 in Cerebral Amyloid Angiopathy and Alzheimer's disease
National Institute on AgingUniversity of Texas Hlth Sci Ctr HoustonUS$654.1k2026–2030
The role of brain border-associated macrophages in aging and cerebral amyloid angiopathy
National Institute on AgingNew York University School of MedicineUS$636.8k2022–2026
Impact of the gut microbiome on B cell-mediated neuroinflammation in cerebral amyloid angiopathy
National Institute on AgingUniversity of Texas Hlth Sci Ctr HoustonUS$696.9k2025–2029
National Heart, Lung, and Blood InstituteVitalantUS$131.3k2024–2026
Vascular Pathology in Early and Asymptomatic Cerebral Amyloid Angiopathy
National Institute on AgingMassachusetts General HospitalUS$785.5k2014–2027
Vascular-immune mechanisms of cerebral amyloid angiopathy and Alzheimer's pathology
National Institute of Neurological Disorders and StrokeJ. David Gladstone InstitutesUS$5.4m2024–2027
VCID CWOW: Identifying Novel Targets to Treat Cerebral Amyloid Angiopathy
National Institute of Neurological Disorders and StrokeThe Jackson LaboratoryUS$5.9m2024–2027
Over a median 5.1 years, bleeding-related lesions appeared in 33.7% of participants with severe enlargement versus 13.6% without severe enlargement.Over a median 5.1 years, bleeding-related lesions appeared in 33.7% of participants with severe enlargement versus 13.6% without severe enlargement.
In a study of App SAA knock-in mice from 4 to 19 months, animals developed progressive amyloid pathology, neuroinflammation, biomarker shifts, and synaptic loss.In a study of App SAA knock-in mice from 4 to 19 months, animals developed progressive amyloid pathology, neuroinflammation, biomarker shifts, and synaptic loss.
In an autopsy study of 265 decedents, vessel mineralization appeared in 58.3% of globus pallidus samples, 28.6% of posterior hippocampus samples, and 13.8% of putamen…In an autopsy study of 265 decedents, vessel mineralization appeared in 58.3% of globus pallidus samples, 28.6% of posterior hippocampus samples, and 13.8% of putamen samples.
Lewy body disease prevalence rose from 0.7% in individuals younger than 50 to 20.0% in those aged 90 and older across 2,480 autopsied human brains.Lewy body disease prevalence rose from 0.7% in individuals younger than 50 to 20.0% in those aged 90 and older across 2,480 autopsied human brains.