Abundance

Biomarker

1 paper studies it1,999 more mention it3 findings

  • In silico1

Upstream

3

Clostridium clostridioforme

upin humans1

1 study
  1. Clostridium clostridioforme increases abundance in humans.

    “we found that Finegoldia magna, Bifidobacterium dentium, and Clostridium clostridioforme had increased abundance in the elderly.”

    Humans

    Human gut microbiome aging clocks based on taxonomic and functional signatures through multi-view learning

    Gut microbes

Bifidobacterium dentium

upin humans1

1 study
  1. Bifidobacterium dentium increases abundance in humans.

    “we found that Finegoldia magna, Bifidobacterium dentium, and Clostridium clostridioforme had increased abundance in the elderly.”

    Humans

    Human gut microbiome aging clocks based on taxonomic and functional signatures through multi-view learning

    Gut microbes

Finegoldia magna

upin humans1

1 study
  1. Finegoldia magna increases abundance in humans.

    “we found that Finegoldia magna, Bifidobacterium dentium, and Clostridium clostridioforme had increased abundance in the elderly.”

    Humans

    Human gut microbiome aging clocks based on taxonomic and functional signatures through multi-view learning

    Gut microbes

Latest

Nov 2025: 31Dec 2025: 86Jan 2026: 58Feb 2026: 36Mar 2026: 39Apr 2026: 36May 2026: 32Jun 2026: 56Jul 2026: 56Aug 2026: 75Sep 2026: 94Oct 2026: 44643 papers in 12 months
bioRxiv

Free testosterone linked to muscle gene activity, not strength

In 81 healthy males aged 18–80, an observational study linked free testosterone to muscle gene activity, but not muscle mass or strength.In 81 healthy males aged 18–80, an observational study linked free testosterone to muscle gene activity, but not muscle mass or strength.

Preprint: cross-sectional · 81 peopleMuscle
Archives of Gerontology and Geriatrics

A pathway may link liver protein clumps and senescence in mice

Experiments in two mouse models of liver disease found protein clumps alongside signs of senescence, a state in which cells stop dividing.Experiments in two mouse models of liver disease found protein clumps alongside signs of senescence, a state in which cells stop dividing.