SLC25A5
4 papers study it4 findings
- 1 lab in 1 country
- Funders: Chonnam National University and 4 more
- Humans1
- Animals2
- Cells1
Effects
4Mitophagy
upin human cells1
1 study
Mitophagy
upin human cells1
SLC25A5 induces mitophagy in human cells.
“ANT2 overexpression increased the adenosine triphosphate production rate by activating glycolysis and induced mitophagy”
Cellsin replicative senescent cells
The Journal of investigative dermatology
Glycolysis
upin human cells1
1 study
Glycolysis
upin human cells1
SLC25A5 activates glycolysis in human cells.
“ANT2 overexpression increased the adenosine triphosphate production rate by activating glycolysis”
Cellsin replicative senescent cells
The Journal of investigative dermatology
Cell proliferation
upin human cells1
1 study
Cell proliferation
upin human cells1
SLC25A5 increases cell proliferation in human cells.
“upregulation of ANT2 in replicative senescent human diploid dermal fibroblasts induced their proliferation and migration”
Cellsreplicative senescencedermal fibroblasts
The Journal of investigative dermatology
Wound healing
upin mice1
1 study
Wound healing
upin mice1
SLC25A5 improves wound healing in mice.
“overexpression of ANT2 in aged mouse skin accelerated the healing of full-thickness cutaneous wounds”
Animalsfull-thickness cutaneous woundsskin
The Journal of investigative dermatology
Latest
Molecular Oncology
Determination of ADP/ATP translocase isoform ratios in malignancy and cellular senescence
Experiments in human cellsCellular senescence
American Journal of Respiratory Cell and Molecular Biology
Loss of ANT1 Increases Fibrosis and Epithelial Cell Senescence in Idiopathic Pulmonary Fibrosis
Experiments in miceCellular senescence
The Journal of Investigative Dermatology
ANT2 Accelerates Cutaneous Wound Healing in Aged Skin by Regulating Energy Homeostasis and Inflammation
Experiments in miceSkin
bioRxiv
Loss of ANT1 Increases Fibrosis and Epithelial Cell Senescence in Idiopathic Pulmonary Fibrosis
Preprint: study in peopleCellular senescence