AgingResearch.NewsJoin free

MIR326

Gene

2 papers3 findings

C, W, M, H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.

Labs are independent when their papers share no last author and no institution.

Includes the processes named after it and trials of drugs aimed at it.

  • 2 independent labs in 1 country
  • Funders: National Natural Science Foundation of China and 3 more
  • Animals1
  • Cells1

Effects

3

Contractile function

downin mouse cells1

1 study

  1. MIR326 impairs contractile function in mouse cells.

    “resulted in impaired mitochondrial and contractile function in cardiomyocytes”

    Cellsin cardiomyocytescardiomyocytes

    Secretion of miRNA-326-3p by senescent adipose exacerbates myocardial metabolism in diabetic mice

    Journal of translational medicineJun 21, 2022

RICTOR

downin mouse cells1

1 study

  1. MIR326 inhibits RICTOR in mouse cells.

    “miRNA-326-3p inhibited the expression of Rictor”

    Cellsin cardiomyocytescardiomyocytes

    Secretion of miRNA-326-3p by senescent adipose exacerbates myocardial metabolism in diabetic mice

    Journal of translational medicineJun 21, 2022

More on RICTOR

Cellular senescence

upin mouse cells1

1 study

  1. MIR326 induces cellular senescence in mouse cells.

    “miR-326-3p mimics accelerated senescence and apoptosis of skin fibroblasts, while miR-326-3p inhibitor could dramatically delay skin fibroblasts senescence and apoptosis.”

    Cellsskin

    Endothelial Cell-Derived Extracellular Vesicles Target TLR4 via miRNA-326-3p to Regulate Skin Fibroblasts Senescence

    Journal of immunology researchMay 18, 2022

More on Cellular senescence

Latest

Related