CAMKK1/STIM1
Gene1 paper1 finding
C · W · M · H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.
Labs are independent when their papers share no last author and no institution.
Against: findings that point the other way from most findings on the same outcome.
No results posted: trials that ended more than a year ago with no results on ClinicalTrials.gov and no paper in ARN’s record.
Company ties: findings with an author at a company, or from a paper that declares the maker’s interest.
A gene’s or a process’s line counts the gene with the processes named after it, as NLRP3 with the NLRP3 inflammasome, and the trials of drugs aimed at it.
- Cells1
Effects
1Apoptosis
upin cells1
1 study
Apoptosis
upin cells1
CAMKK1/STIM1 induces apoptosis in cells.
Cellsforced CAMKK1/STIM1 co-overexpressionosteoclast
Deleting a signaling protein limits bone loss in mice · Pharmacological research · 8 Oct 2026
Latest
1Deleting a signaling protein limits bone loss in mice
In mice lacking ovaries to mimic estrogen loss, deleting the Camkk1 gene from bone-degrading cells attenuated the loss of spongy bone.In mice lacking ovaries to mimic estrogen loss, deleting the Camkk1 gene from bone-degrading cells attenuated the loss of spongy bone.