Childhood BMI was not clearly linked with carotid plaque in humans (Genetically predicted childhood BMI; adult outcome) (β = −0.095 (95% CI −0.264 to 0.073) per 1-standard deviation increase in childhood BMI).
“but not CP (β = −0.095; 95% CI, −0.264 to 0.073)”
Childhood BMI is associated with coronary artery calcification in humans (Genetically predicted childhood BMI; adult outcome) (β = 0.114 (95% CI 0.032–0.196) per 1-standard deviation increase in childhood BMI).
“childhood BMI was associated with higher coronary PB (β = 0.326; 95% CI, 0.142–0.511) and CAC (β = 0.114; 95% CI, 0.032–0.196)”
Childhood BMI is associated with coronary plaque burden in humans (Genetically predicted childhood BMI; adult outcome) (β = 0.326 (95% CI 0.142–0.511) per 1-standard deviation increase in childhood BMI).
“A genetically predicted 1-standard deviation increase in childhood BMI was associated with higher coronary PB (β = 0.326; 95% CI, 0.142–0.511)”
Genetic analysis linked higher childhood body mass index to adult heart-artery plaque and calcium buildup, with association estimates of 0.326 and 0.114, respectively.Genetic analysis linked higher childhood body mass index to adult heart-artery plaque and calcium buildup, with association estimates of 0.326 and 0.114, respectively.
In FinnGen, genetically predicted hip and thigh fat, adjusted for body size, was associated with lower liver cancer odds, with an odds ratio of 0.444.In FinnGen, genetically predicted hip and thigh fat, adjusted for body size, was associated with lower liver cancer odds, with an odds ratio of 0.444.