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Streptococcus

Exposure

1 paper2 findings

C, W, M, H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.

Labs are independent when their papers share no last author and no institution.

Against: findings that point the other way from most findings on the same outcome.

On aging: the drug’s trials ARN judged to be about aging, of every trial that tests it for any use.

No results posted: trials that ended more than a year ago with no results on ClinicalTrials.gov and no paper in ARN’s record.

Company ties: findings with an author at a company, or from a paper whose competing-interests statement declares ties to one.

  • 1 lab in 1 country
  • Funders: National Natural Science Foundation of China and 1 more
  • Humans1

Outcomes

2

Lifespan

downin humans1

1 study

  1. Streptococcus shortens lifespan in humans (β = -0.0247).

    “Streptococcus was causally associated with aging (β = 0.3310, P = 0.0019) and lifespan-shortening (β = - 0.0247, P = 0.0184)”

    HumansGenetically inferred association using Mendelian randomization

    Identification of gut microbiota causally associated with aging and longevity and mediation roles of the cerebrospinal fluid proteins: proteomic genetic evidence from Mendelian randomization

    GeroScience23 Jul 2025

Aging-related phenotype

upin humans1

1 study

  1. Streptococcus increases aging-related phenotype in humans (β = 0.3310).

    “Streptococcus was causally associated with aging (β = 0.3310, P = 0.0019)”

    HumansGenetically inferred association using Mendelian randomization

    Identification of gut microbiota causally associated with aging and longevity and mediation roles of the cerebrospinal fluid proteins: proteomic genetic evidence from Mendelian randomization

    GeroScience23 Jul 2025

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