Healthy tendon stem cell-derived exosomes

Healthy tendon stem cell-derived exosomes has been studied in 1 paper since 2024: none in people yet, 1 in animals. The research so far is in animals. The newest paper, Mar 8, 2024: “Healthy Tendon Stem Cell-Derived Exosomes Promote Tendon-To-Bone Healing of Aged Chronic Rotator Cuff Tears by Breaking the Positive-Feedback Cross-Talk between Senescent Tendon Stem Cells and Macrophages through…”.

Intervention

1 paper studies it2 findingsUpdated

None of its findings points the other way.

  • 1 lab in 1 country
  • Funder: National Natural Science Foundation of China
  • Animals1

What did studies of Healthy tendon stem cell-derived exosomes find?

2

Studies of Healthy tendon stem cell-derived exosomes measured 2 outcomes; the most studied are inflammation (1 study) and tendon-to-bone healing (1).

Inflammation

downin rats1

1 study
  1. Healthy tendon stem cell-derived exosomes decrease inflammation in rats.

    “h-TSC-Exos can shift the microenvironment in the tendon-to-bone interface from a pro-inflammatory to an anti-inflammatory type at the acute postoperative stage”

    Animalsaged-chronic rotator cuff tear rat modeltendon-to-bone interface

    Healthy Tendon Stem Cell-Derived Exosomes Promote Tendon-To-Bone Healing of Aged Chronic Rotator Cuff Tears by Breaking the Positive-Feedback Cross-Talk between Senescent Tendon Stem Cells and Macrophages through the Modulation of Macrophage Polarization

    Small (Weinheim an der Bergstrasse, Germany)

More on Inflammation

Tendon-to-bone healing

upin rats1

1 study
  1. Healthy tendon stem cell-derived exosomes improve tendon-to-bone healing in rats.

    “improve the tendon-to-bone healing results, which are associated with the rejuvenated s-TSCs.”

    Animalsaged-chronic rotator cuff teartendon-to-bone interface

    Healthy Tendon Stem Cell-Derived Exosomes Promote Tendon-To-Bone Healing of Aged Chronic Rotator Cuff Tears by Breaking the Positive-Feedback Cross-Talk between Senescent Tendon Stem Cells and Macrophages through the Modulation of Macrophage Polarization

    Small (Weinheim an der Bergstrasse, Germany)

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