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SLC30A4-AS1

Gene

1 paper2 findings

Taxon

C, W, M, H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.

Includes the processes named after it and trials of drugs aimed at it.

  • 1 lab in 1 country
  • Funders: China Postdoctoral Science Foundation and 2 more
  • Humans1

Effects

2

TP53BP1 alternative splicing

mediatesin human cells1

1 study

  1. SLC30A4-AS1 mediates TP53BP1 alternative splicing in human cells.

    “SLC30A4-AS1 interacted with SRSF3 to affect the alternative splicing (AS) of TP53BP1 and alter the expression of TP53BP1-204.”

    Cellsinflammatory environments; interaction with SRSF3periodontal ligament stem cell

    SLC30A4-AS1 Mediates the Senescence of Periodontal Ligament Stem Cells in Inflammatory Environments via the Alternative Splicing of TP53BP1

    Cell proliferation21 Nov 2024

Cellular senescence

upin human cells1

1 study

  1. SLC30A4-AS1 induces cellular senescence in human cells.

    “SLC30A4-AS1 overexpression-induced PDLSC senescence was abolished by TP53BP1-204 knockdown.”

    CellsSLC30A4-AS1 overexpression in inflammatory environmentsperiodontal ligament stem cell

    SLC30A4-AS1 Mediates the Senescence of Periodontal Ligament Stem Cells in Inflammatory Environments via the Alternative Splicing of TP53BP1

    Cell proliferation21 Nov 2024

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