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Hey

There is 1 paper on Hey in aging research: none in people yet, 1 in animals.

Gene

1 paper studies it2 findings

Taxon

See all 1

None of its findings points the other way.

C, W, M, H: cells, worms or flies, mice, humans. Dark: independent labs found the same change there. Light: one lab or one study. Outline: nothing yet.

Includes the processes named after it and trials of drugs aimed at it.

  • 1 lab in 1 country
  • Funders: Deutsche Forschungsgemeinschaft and 2 more
  • Model organisms1

Effects

2

Aging phenotypes

downin flies1

1 study

  1. Hey decreases aging phenotypes in flies (continued Hey expression in enterocytes).

    “aging phenotypes were suppressed by continued expression of Hey in ECs”

    Model organismscontinued Hey expression in enterocytesmidgut

    The transcription factor Hey and nuclear lamins specify and maintain cell identity

    eLife

Enterocyte identity and nuclear organization

is required forin flies1

1 study

  1. Hey is required for enterocyte identity and nuclear organization in flies (Hey-deficient enterocytes).

    “Lineage tracing established that Hey-deficient ECs are unable to maintain their unique nuclear organization and identity”

    Model organismsHey-deficient enterocytesmidgut

    The transcription factor Hey and nuclear lamins specify and maintain cell identity

    eLife

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