G3-I
G3-I has been studied in 1 paper since 2020: none in people yet, 1 in animals. The research so far is in animals. The newest paper, Oct 21, 2020: “Mammalian-unique eIF4E2 maintains GSK3β proline kinase activity to resist senescence against hypoxia”.
1 paper studies it1 findingUpdated
- 1 lab in 1 country
- Animals1
What did studies of G3-I find?
1Studies of G3-I measured 1 outcome; the most studied are cellular senescence (1 study).
Cellular senescence
upin cells1
1 study
Cellular senescence
upin cells1
G3-I induces cellular senescence in cells (peptide e2-I or G3-I).
“Peptide e2-I or G3-I that disrupts this pathway dephosphorylates p53 at multiple S/T-P, which accelerate senescence”
Cellspeptide e2-I or G3-I
Mammalian-unique eIF4E2 maintains GSK3β proline kinase activity to resist senescence against hypoxia
bioRxiv (Cold Spring Harbor Laboratory)Preprint
Latest
bioRxiv
Mammalian-unique eIF4E2 maintains GSK3β proline kinase activity to resist senescence against hypoxia
Preprint: experiments in miceCellular senescence