EPHB2

EPHB2 has been studied in 1 paper since 2016: 1 observational study in people. The newest paper, Jan 6, 2016: “Activation of EphA4 and EphB2 Reverse Signaling Restores the Age-Associated Reduction of Self-Renewal, Migration, and Actin Turnover in Human Tendon Stem/Progenitor Cells”.

Intervention

1 paper studies it2 findingsUpdated

  • 1 lab in 1 country
  • Funders: Deutsche Forschungsgemeinschaft and 1 more
  • Humans1

What did studies of EPHB2 find?

2

Studies of EPHB2 measured 2 outcomes; the most studied are actin turnover (1 study) and cell migration (1).

Actin turnover

upin human cells1

1 study
  1. EPHB2 improves actin turnover in human cells.

    “activation of EphA4- or EphB2-dependent reverse signaling could restore the migratory ability and normalize the actin turnover of A-TSPC.”

    Cellsaged tendon stem/progenitor cellsclustered proteintendon stem/progenitor cell

    Activation of EphA4 and EphB2 Reverse Signaling Restores the Age-Associated Reduction of Self-Renewal, Migration, and Actin Turnover in Human Tendon Stem/Progenitor Cells

    Frontiers in aging neuroscience

Cell migration

upin human cells1

1 study
  1. EPHB2 improves cell migration in human cells.

    “activation of EphA4- or EphB2-dependent reverse signaling could restore the migratory ability and normalize the actin turnover of A-TSPC.”

    Cellsaged tendon stem/progenitor cellsclustered proteintendon stem/progenitor cell

    Activation of EphA4 and EphB2 Reverse Signaling Restores the Age-Associated Reduction of Self-Renewal, Migration, and Actin Turnover in Human Tendon Stem/Progenitor Cells

    Frontiers in aging neuroscience

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