Papers by Ding Shen

Biochemical Pharmacology26 Sep
Sephin1 promotes mitophagy and reduces alpha-synuclein toxicity in Parkinson models
The compound directly binds Prohibitin-2 to activate PINK1-PRKN-dependent clearance of damaged mitochondria, easing motor deficits in transgenic mice.The compound directly binds Prohibitin-2 to activate PINK1-PRKN-dependent clearance of damaged mitochondria, easing motor deficits in transgenic mice.
Experiments in human cellsParkinson's disease
Paper
ACS Applied Materials & Interfaces17 Aug
Near-Infrared Region I-Responsive Composite Scaffolds Promote Bone Defect Repair by Jointly Regulating Angiogenic and Osteogenic Differentiation through Magnesium Ions and Metformin
Experiments in animalsInterventions
Paper